Characterization of twenty-five ovarian tumour cell lines that phenocopy primary tumours

نویسندگان

  • Tan A Ince
  • Aurea D Sousa
  • Michelle A Jones
  • J Chuck Harrell
  • Elin S Agoston
  • Marit Krohn
  • Laura M Selfors
  • Wenbin Liu
  • Ken Chen
  • Mao Yong
  • Peter Buchwald
  • Bin Wang
  • Katherine S Hale
  • Evan Cohick
  • Petra Sergent
  • Abigail Witt
  • Zhanna Kozhekbaeva
  • Sizhen Gao
  • Agoston T Agoston
  • Melissa A Merritt
  • Rosemary Foster
  • Bo R Rueda
  • Christopher P Crum
  • Joan S Brugge
  • Gordon B Mills
چکیده

Currently available human tumour cell line panels consist of a small number of lines in each lineage that generally fail to retain the phenotype of the original patient tumour. Here we develop a cell culture medium that enables us to routinely establish cell lines from diverse subtypes of human ovarian cancers with >95% efficiency. Importantly, the 25 new ovarian tumour cell lines described here retain the genomic landscape, histopathology and molecular features of the original tumours. Furthermore, the molecular profile and drug response of these cell lines correlate with distinct groups of primary tumours with different outcomes. Thus, tumour cell lines derived using this methodology represent a significantly improved platform to study human tumour pathophysiology and response to therapy.

برای دانلود رایگان متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

منابع مشابه

Evaluating cell lines as tumour models by comparison of genomic profiles

Cancer cell lines are frequently used as in vitro tumour models. Recent molecular profiles of hundreds of cell lines from The Cancer Cell Line Encyclopedia and thousands of tumour samples from the Cancer Genome Atlas now allow a systematic genomic comparison of cell lines and tumours. Here we analyse a panel of 47 ovarian cancer cell lines and identify those that have the highest genetic simila...

متن کامل

NuMA Overexpression in Epithelial Ovarian Cancer

Highly aneuploid tumours are common in epithelial ovarian cancers (EOC). We investigated whether NuMA expression was associated with this phenomenon.NuMA protein levels in normal and tumour tissues, ovarian cell lines and primary cultures of malignant cells derived from ovarian ascitic fluids were analysed by Affymetrix microarray analysis, immunoblotting, immunohistochemistry (IHC) and immunof...

متن کامل

Value of A103 (melan-A) immunostaining in the differential diagnosis of ovarian sex cord stromal tumours.

AIMS To assess A103 (melan-A) immunoreactivity in a range of ovarian sex cord stromal tumours and to evaluate it for the differential diagnosis of other neoplasms. METHODS Paraffin embedded tissue sections from 45 sex cord stromal tumours and 44 potential histological mimics were examined immunohistochemically using the antibody A103. The sex cord stromal group included 21 adult granulosa cel...

متن کامل

Value of A103 (melan-A) immunostaining in the diVerential diagnosis of ovarian sex cord stromal tumours

Aims—To assess A103 (melan-A) immunoreactivity in a range of ovarian sex cord stromal tumours and to evaluate it for the diVerential diagnosis of other neoplasms. Methods—ParaYn embedded tissue sections from 45 sex cord stromal tumours and 44 potential histological mimics were examined immunohistochemically using the antibody A103. The sex cord stromal group included 21 adult granulosa cell tum...

متن کامل

ABT-627, a potent endothelin receptor A antagonist, inhibits ovarian carcinoma growth in vitro.

Endothelin-1 (ET-1) is present at high concentrations in ovarian cancer ascites and is overexpressed in primary and metastatic ovarian carcinomas. In these tumours the presence of ET-1 is associated with enhanced neovascularization and with vascular endothelial growth factor (VEGF) expression. In these tumour cells, ET-1 acts as an autocrine growth factor selectively through the receptor ET(A),...

متن کامل

ذخیره در منابع من


  با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید

عنوان ژورنال:

دوره 6  شماره 

صفحات  -

تاریخ انتشار 2015